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Gait Disorders, Imbalance, and Falls

Chapter 28 | Part 2: Cardinal Manifestations and Presentation of Diseases · Part 2 – Cardinal Manifestations & Presentation · Chapter 28


Key Clinical Points

  1. Balance requires integration of visual, vestibular, and proprioceptive inputs; loss of any two pathways compromises stability.
  2. Prevalence of gait disorders is high in the elderly (15% in >65 years; ~40% in >85 years).
  3. Frontal gait disorder (higher-level) is common in the elderly, often caused by vascular disease or NPH.
  4. Parkinsonian gait features include stooped posture, shuffling, decreased arm swing, festination, and freezing.
  5. Sensory ataxia is characterized by a positive Romberg sign, worsening in the dark, and looking at feet while walking.
  6. Cerebellar ataxia presents with wide-based gait, truncal sway, and late falls.
  7. Fall risk factors include intrinsic (vision, gait, weakness) and extrinsic (environment); polypharmacy is a major modifiable risk.
  8. TUG test >12 seconds indicates high risk for falls.
  9. Functional gait disorder presents with sudden onset, inconsistent deficits, and improvement with distraction.
  10. Hip fractures from falls significantly increase mortality risk and lead to potential nursing home admission.

DEFINITION & CLASSIFICATION

Balance: Ability to maintain equilibrium—a dynamic state where center of mass is controlled relative to lower extremities, gravity, and support surface despite perturbations. • Sensory Inputs: Primarily generated by visual, vestibular, and proprioceptive systems (muscle spindles/joints). • Redundancy: Healthy redundancy exists; loss of 2 out of 3 pathways → compromised standing balance. • Mechanism: ◦ Brainstem, cerebellum, and forebrain modify spinal pattern generators to promote stepping. ◦ Step generation in primates depends on pontine tegmentum, midbrain, and subthalamic region. ◦ Reticular formation and descending pathways (ventromedial spinal cord) execute locomotor synergies. ◦ Cerebral control provides goal/purpose, obstacle avoidance, and adaptation to terrain. • Postural Control: Requires maintaining center of mass over base of support; includes long latency responses (starting 110 milliseconds after perturbation). • Failure Points: Can occur at any level → presents as difficulty maintaining posture while standing or walking.

Anatomy and Physiology of Gait

Complexity: Bipedal walking is complex; even minor neurologic deficits can compromise performance. • Clinical Observation: Gait disorders are often viewed as a product of a neurologic deficit combined with functional adaptation. • Adaptation: Unique features of failing gait are often masked by adaptive responses to threatened balance.


EPIDEMIOLOGY

Prevalence: ◦ Age >65: 15% have gait disorders. ◦ Age ≥80: 1 in 4 use a mechanical aid for ambulation. ◦ Age ≥85: Prevalence approaches 40%. • Fall Risk: ◦ Over 1/3 of people >65 living in the community fall annually (higher rate in nursing homes/hospitals). ◦ Impact: Hip fractures → hospitalization, potential nursing home admission, and increased mortality risk. ◦ Secondary Risks: Dehydration, rhabdomyolysis (from prolonged time on ground), and brain/spinal injury. • Psychological Impact: 1 in 5 elderly individuals restrict activity due to fear of falling; loss of ambulation reduces quality of life.


ETIOLOGY & PATHOPHYSIOLOGY

Non-neurologic causes: Antalgic gait (osteoarthritis), asymmetry (contractures/orthopedic deformities), and impaired vision. • Neurologic Gait Disorders:Sensory ataxic: Peripheral sensory neuropathy. ◦ Parkinsonian: Parkinson's disease, drug-induced parkinsonism, dementia with parkinsonism. ◦ Higher level: Vascular encephalopathy, normal pressure hydrocephalus (NPH), severe dementia, hypoxic ischemic encephalopathy. ◦ Cerebellar ataxic: Cerebellar stroke, MS, essential tremor, postvaccinal cerebellitis, chronic alcohol abuse, multiple system atrophy. ◦ Cautious: Idiopathic, associated fear of falling. ◦ Paretic/hypotonic: Neurogenic claudication, diabetic neuropathy, trauma/surgery injury, GBS. ◦ Spastic: Ischemic stroke, intracerebral hemorrhage, congenital. ◦ Vestibular ataxic: Bilateral vestibulopathy, vestibular neuronitis, Ménière's attack, acoustic neuroma. ◦ Dyskinetic: Levodopa-induced dyskinesia, chorea.

Classification of Gait Disorders

Descriptive Classification: Based on abnormal physiology and biomechanics (e.g., sensory ataxic, parkinsonian). ◦ Note: Many failing gaits look similar due to common patterns of adaptation to threatened balance.

Prevalence of Neurologic Gait Disorders

Table 28-1 provides the breakdown of cases: • Sensory ataxic: 18% (46 total) → Peripheral sensory neuropathy. • Parkinsonian: 16% (34 total) → PD, drug-induced, dementia with parkinsonism. • Higher level: 8% (31 total) → Vascular encephalopathy (20), NPH (1), severe dementia (7). • Cerebellar ataxic: 6% (10 total) → Stroke, MS, essential tremor, alcohol abuse. • Cautious: 6% (7 total) → Idiopathic/fear of falling. • Paretic/hypotonic: 5% (14 total) → Neurogenic claudication, diabetic neuropathy, trauma. • Spastic: 5% (7 total) → Stroke, hemorrhage. • Vestibular ataxic: 3% (6 total) → Vestibulopathy, neuronitis, Ménière's. • Dyskinetic: 1% (4 total) → Levodopa-induced dyskinesia, chorea.


CLINICAL FEATURES

General Observations: Gait is primarily assessed via observation; cadence, velocity, and stride length can be measured. • Parkinsonian Features: ◦ Stooped posture, shuffling gait, decreased arm swing, festination, and freezing of gait. ◦ Differentiation: ◦ PSP: More erect posture than PD; falls within first year suggest PSP. ◦ Vascular Parkinsonism: Broad-based/shuffling with reduced arm swing; disproportionate involvement of gait early in course. • Frontal Gait Disorder (Higher-level): ◦ Characteristics: Shuffling, freezing, wide base, short stride, difficulty with starts and turns. ◦ 'Slipping clutch' syndrome: Difficulty with gait initiation. ◦ Etiology: Most common cause is vascular disease (subcortical small-vessel). ◦ NPH: Presents with similar gait; MRI shows ventricular enlargement and periventricular white matter change. • Cerebellar Gait Ataxia: ◦ Characteristics: Wide base, truncal sway, erratic foot placement, difficulty turning. ◦ Clinical signs: Inability to walk tandem heel-to-heel. • Sensory Ataxia: ◦ Features: Romberg positive, worse in dark, patient looks down at feet. ◦ Mechanism: Loss of proprioception requires active visual monitoring. • Vestibular Disorders: ◦ Symptoms: Vertigo (subjective), nystagmus, impaired standing balance. ◦ Ototoxic drugs: May lack vertigo/nystagmus but have impaired balance and difficulty in the dark. • Neuromuscular Disease: ◦ Steppage gait: Increased step height to compensate for foot drop (peripheral neuropathy). ◦ Myopathy/Dystrophy: Proximal weakness → excess pelvic sway. • Functional Gait Disorder: ◦ Features: Sudden onset, inconsistent deficits, waxing/waning, improvement with distraction. ◦ Presentation: Slow motion, overcautious gait, astasia-abasia, bouncing. • Other Disorders: ◦ Orthostatic tremor: High-frequency, low-amplitude; worse on standing. ◦ Lumbar spinal stenosis: Stooped posture to alleviate pain (mimics early parkinsonism). ◦ Dyskinetic: Dancing quality (Huntington's), stereotypic patterns (Tardive dyskinesia).

Comparative Features of Ataxias

Table 28-2 summarizes the differences between Cerebellar, Sensory, and Frontal Gait: • Cerebellar: Wide-based; Irregular/lurching stride; Abnormal heel → shin; Unsteady turns; Late falls. • Sensory: Wide-based (looks down); Regular stride with path deviation; ± Romberg; Frequent falls. • Frontal: Wide-based; Short, shuffling stride; Normal heel → shin; Hesitant/multistep turns; Frequent falls.


DIFFERENTIAL DIAGNOSIS

By Etiology: ◦ Neurologic (Parkinsonian, Cerebellar, etc.) vs. Non-neurologic (Pain/Arthritic, Vision, Vestibular). • By Physiology and Biomechanics: ◦ Distinction between motor control issues (Frontal), sensory deficits (Sensory Ataxia), and vestibular dysfunction.


DIAGNOSTIC APPROACH

  1. History Taking: ◦ Identify fall patterns: Sudden drop → suspect syncope, seizure, or neuro event; Tripping/slipping → mechanical/environmental. ◦ Assess prodromal symptoms: Dizziness, vertigo, presyncopal symptoms, focal weakness. ◦ Review medications: Benzodiazepines, opioids, antipsychotics, antiepileptics, antidepressants, antiarrhythmics, diuretics. ◦ Determine duration and consistency of gait issues (Sudden/inconsistent → Functional).
  2. Physical Examination: ◦ Observe gait parameters: Cadence, velocity, stride length. ◦ Perform Romberg test to assess sensory/vestibular input. ◦ Assess for 'slipping clutch' or festination (Parkinsonian signs). ◦ Conduct cardiac examination (especially in patients with sudden falls).
  3. Diagnostic Tests: ◦ MRI: Sensitive for cerebral lesions (vascular, demyelinating) and screening for hydrocephalus. ◦ Lumbar puncture/dynamic test: Required to confirm NPH.

MANAGEMENT & TREATMENT

  1. Pharmacologic Management: ◦ Dopaminergic drugs: Optimize for patients with freezing of gait. ◦ MAO-B inhibitors: Use rasagiline or selegiline to improve end-of-dose gait freezing.
  2. Non-Pharmacologic Management: ◦ Instrumentation: Use of instrumented walkways for quantitative analysis. ◦ Cueing: Implementation of auditory and visual cueing strategies to overcome freezing. ◦ Environmental modification: Address extrinsic factors like wet surfaces or improper footwear.

COMPLICATIONS & PROGNOSIS

Fall Consequences: ◦ Physical injury (fractures, head trauma). ◦ Reduced mobility → loss of independence. ◦ Increased mortality risk (especially from hip fractures). • Quality of Life: ◦ Fear of falling leads to self-imposed activity restriction. ◦ Loss of ambulation correlates with increased morbidity and mortality.


SPECIAL POPULATIONS

Cognitive Impairment: Patients with dementia are at higher risk for falls and injury. • Metabolic/Toxic Disorders: ◦ Chronic renal disease or hepatic failure → asterixis → impaired postural support. ◦ Drug toxicity: Neuroleptics and long-acting benzodiazepines significantly increase fall risk.


KEY PEARLS & HIGH-YIELD POINTS

TUG Test: Score >12 seconds → high risk for falls. • Frontal Gait: Often a 'higher-level' motor control disorder, not an apraxia; most common cause is subcortical vascular disease. • Sensory Ataxia: Characterized by the need for visual compensation (looking at feet) and failure in dark/closed eyes. • Cerebellar Ataxia: Falls are typically a 'late' event as patients use postural compensation early on. • Risk Factors Table: Table 28-3 lists key risk factors: History of falls, Gait/balance disorder, Visual deficits, Physical disability (weakness, OA), Orthostatic hypotension, Depression, Cognitive impairment, and Polypharmacy.


Reference Tables

TABLE 28-1 Prevalence of Neurologic Gait Disorders

Harrison's 22e, p.177

NEUROLOGIC GAIT DISORDER NO. (%)a TOTAL NUMBERb CAUSES (NO.)
Single neurologic gait disorder 81 (69%)
Sensory ataxic 22 (18%) 46 Peripheral sensory neuropathy (46)
Parkinsonian 19 (16%) 34 Parkinson’s disease (18), drug-induced parkinsonism (8), dementia with parkinsonism (4),
parkinsonism (4)
Higher level 9 (8%) 31 Vascular encephalopathy (20), normal pressure hydrocephalus (1), severe dementia (7),
hypoxic ischemic encephalopathy (1), unknown (1)
Cerebellar ataxic 7 (6%) 10 Cerebellar stroke (3), cerebellar lesion due to multiple sclerosis (1), severe essential tremor
(3), postvaccinal cerebellitis (1), chronic alcohol abuse (1), multiple system atrophy (1)
Cautious 7 (6%) 7 Idiopathic, associated fear of falling (7)
Paretic/hypotonic 6 (5%) 14 Neurogenic claudication (7), diabetic neuropathy (1), nerve lesion due to trauma or surgery
(4), distal paraparesis after Guillain-Barré syndrome (1), unknown (2)
Spastic 6 (5%) 7 Ischemic stroke (3), intracerebral hemorrhage (3), congenital (1)
Vestibular ataxic 4 (3%) 6 Bilateral vestibulopathy (3), recent vestibular neuronitis (1), recent Ménière’s attack (1),
acoustic neuroma with surgery (1)
Dyskinetic 1 (1%) 4 Levodopa-induced dyskinesia (3), chorea (1)
36 (30%)
Total 117

TABLE 28-2 Features of Cerebellar Ataxia, Sensory Ataxia, and Frontal Gait Disorders FEATURE Base of support Velocity…

Harrison's 22e, p.179

FEATURE CEREBELLAR ATAXIA SENSORY ATAXIA FRONTAL GAIT
Base of support Wide-based Wide-based, looks down Wide-based
Variable Slow
Stride Irregular, lurching Regular with path deviation Short, shuffling
+/– Unsteady, falls
Heel → shin Abnormal +/– Normal
Normal Normal
Turns Unsteady +/– Hesitant, multistep
+ +++
Falls Late event Frequent Frequent

TABLE 28-3 Common Risk Factors for Falls in Older Adults

Harrison's 22e, p.180

  • RISK FACTOR
  • History of falls
  • Gait and/or balance disorder
  • Visual deficits
  • Physical disability
    • Includes muscle weakness, use of assistive devices, osteoarthritis of the knee
  • Orthostatic hypotension
  • Depression
  • Cognitive impairment
  • Medications
    • Polypharmacy, use of antipsychotics, antidepressants, benzodiazepines,
    anticholinergics, antihypertensives, and diuretics