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Hypertension

Chapter 288 | Part 1: Disorders of the Cardiovascular System · Part 6 – Cardiovascular Disorders · Chapter 288


Key Clinical Points

  1. Hypertension is defined as an average of three SBP or DBP readings at or above the 95th percentile, or an SBP ≥130 and DBP ≥80 mmHg in adults ≥18 years.
  2. White coat hypertension (high office, normal out-of-office) has a CVD risk profile similar to normotensive individuals; masked hypertension (normal office, high out-of-office) has a risk profile similar to sustained hypertension.
  3. Secondary hypertension should be suspected in cases of new-onset, abrupt worsening, disproportionate target organ damage, or unprovoked hypokalemia/proteinuria/LVH.
  4. Obstructive sleep apnea (OSA) is the most common cause of secondary hypertension.
  5. Nonpharmacologic interventions include the DASH diet (-5 mmHg SBP), weight loss (-1 mmHg per kg), and sodium reduction (25% reduction → -5 mmHg SBP).
  6. Potassium supplementation (+2000 mg/d) is contraindicated in hyperkalemia or advanced kidney disease.
  7. Risk of CVD is significantly higher when hypertension is accompanied by other risk factors (over 30-fold difference in 10-year predicted risk).
  8. Resistant hypertension requires a systematic search for secondary causes and potential escalation to MRA or vasodilators (hydralazine/minoxidil).
  9. Pharmacologic management is tiered based on the severity of elevation above target goals.
  10. Accurate measurement requires specific positioning, such as feet flat on the floor.

DEFINITION & OVERVIEW

Definition: ◦ Average of three SBP or DBP readings at or above the 95th percentile. ◦ Or an SBP ≥130 and DBP ≥80 mmHg in adults ≥18 years. • Measurement Standards: ◦ Use average of two or more readings at two or more visits to estimate usual level. ◦ If SBP and DBP are in different categories, the higher classification is chosen. • Clinical Impact: ◦ Hypertension is a leading risk factor for cardiovascular disease (CVD), including stroke, coronary heart disease (CHD), heart failure (HF), peripheral arterial disease (PAD), chronic kidney disease (CKD)/end-stage kidney disease (ESKD), dementia, and all-cause mortality. • Physiology: ◦ Blood pressure is controlled by cardiac output and peripheral resistance. ◦ Peripheral resistance is determined by vascular structure and vascular function (regulated by RAAS and sympathetic nervous system). ◦ Endothelial dysfunction may play a role in the initiation or progression of hypertension.


EPIDEMIOLOGY

General Trends: ◦ Prevalence increases with age; SBP rises until the 8th decade, DBP until the 5th. ◦ Isolated systolic hypertension is common in older adults due to widened pulse pressure. • U.S. Statistics: ◦ >103 million adults have hypertension. ◦ Prevalence: 20–30% (ages 20–44) → 80–85% (age ≥75). ◦ Racial Disparity: Non-Hispanic black adults have a higher prevalence (59%) and earlier onset/more complications than other groups. • Risk Assessment: ◦ Risk of CVD is significantly higher in older adults due to co-existing risk factors. ◦ There is a >30-fold difference in 10-year predicted risk of CVD for those with multiple risk factors compared to isolated hypertension.


ETIOLOGY & PATHOPHYSIOLOGY

Primary Hypertension: ◦ Most common form; no obvious underlying anatomic cause. ◦ Evidence suggests a heritable component, but genetic studies show only modest polygenic associations. • Secondary Hypertension: ◦ Minority of patients with an overt underlying anatomic or biochemical cause. ◦ Indicators for investigation: 1. Treatment-resistant hypertension 2. Abrupt worsening of hypertension 3. Disproportionate target organ damage for level of BP 4. Laboratory/diagnostic findings (unprovoked hypokalemia, proteinuria, or left ventricular hypertrophy).


CLINICAL FEATURES

Symptoms: ◦ Often asymptomatic; symptoms usually relate to end-organ damage or secondary causes. • Secondary Cause Indicators: ◦ Obstructive sleep apnea (OSA): snoring, poor quality sleep, breathing pauses, and daytime sleepiness. • White Coat Hypertension: ◦ High office BP, normal out-of-office → CVD risk profile similar to normotensive individuals; may be treated with nonpharmacologic interventions. • Masked Hypertension: ◦ Normal office BP, high out-of-office → CVD risk profile similar to sustained hypertension; requires pharmacologic therapy. • Target Organ Damage: ◦ Left ventricular hypertrophy (LVH), proteinuria, and retinopathy.


DIFFERENTIAL DIAGNOSIS

Primary vs. Secondary: ◦ Primary: No anatomic cause; most common. ◦ Secondary: Underlying causes including OSA, renal artery stenosis, primary aldosteronism, pheochromocytoma, Cushing syndrome, and thyroid disorders. • Medication-Induced Hypertension: ◦ Prescription: Amphetamines, antidepressants, antipsychotic agents, decongestants, oral contraceptives, NSAIDs, systemic corticosteroids. ◦ Illicit: Cocaine, marijuana, amphetamines, methylenedioxymethamphetamine. ◦ Herbal/Other: Arnica, ephedra (Ma-Huang), ginseng, guarana, licorice, St. John's wort; caffeine and some anesthetics (short-term).


INVESTIGATIONS & DIAGNOSIS

  1. Initial Evaluation: • Identify target organ damage (LVH, proteinuria), secondary causes (kidney disease, primary aldosteronism), and comorbidities (DM, hyperlipidemia).
  2. Laboratory Panel: ◦ CBC, Serum electrolytes (Na, K, Ca), Serum creatinine/eGFR. ◦ Lipid profile, Glycemic status (HbA1c or fasting glucose). ◦ TSH, Urinalysis, and urine albumin-to-creatinine ratio (UACR).
  3. Diagnostic Procedures: ◦ 12-lead ECG to assess for LVH.
  4. Specialized Assessment: ◦ Evaluate for secondary causes in patients with new-onset, rapid worsening, or disproportionate organ damage.

MANAGEMENT & TREATMENT

  1. Nonpharmacologic Interventions (Table 288-2):DASH Diet: ◦ Expected change: SBP reduction of ~5 mmHg in patients with HTN; 2–3 mmHg in those without. • Weight Loss: ◦ Expected change: SBP reduction of ~1 mmHg per kg of weight loss. • Sodium Reduction: ◦ Target: <1500 to 2300 mg/day. ◦ Expected change: Reduction by ~25% → SBP reduction of ~5 mmHg. • Potassium Supplementation: ◦ Dose: +2000 mg/d (~50 mmol/d). Recommended intake is ~3500 mg/d. ◦ Contraindication: Hyperkalemia, advanced kidney disease, or medications increasing hyperkalemia risk. • Physical Activity: ◦ Recommendation: ≥150 min of aerobic exercise per week. • Alcohol Reduction: ◦ Target: ≤2 standard drinks/day (men), ≤1 (women).
  2. Pharmacologic Treatment (Table 288-3 & Figure 288-4):Initial Selection (Figure 288-4): ◦ Monotherapy: For patients with SBP/DBP close to 130/80 mmHg. ◦ Combination Therapy: For patients with SBP >120 mmHg above treatment goal. ◦ Triple-drug Therapy: For patients with SBP/DBP >130/80 mmHg. • Drug Classes and Dosages:Diuretics (Thiazide/Thiazide-like): Chlorthalidone (12.5–25 mg), Hydrochlorothiazide (12.5–50 mg), Indapamide (1.25–2.5 mg). ◦ ACE Inhibitors: Enalapril (5–40 mg), Lisinopril (10–40 mg), Benazepril (10–40 mg), Ramipril (2.5–20 mg), Trandolapril (1–4 mg). ◦ ARBs: Losartan (50–100 mg), Valsartan (80–320 mg), Azilsartan (40–80 mg), Candesartan (8–32 mg), Olmesartan (20–40 mg). ◦ CCBs: Amlodipine (2.5–10 mg), Felodipine (2.5–10 mg), Nifedipine LA (30–90 mg). ◦ CCB (non-DHP): Diltiazem ER (120–360 mg), Verapamil ER (100–300 mg). ◦ Beta-blockers: Metoprolol (50–200 mg), Carvedilol (20–80 mg), Nebivolol (5–40 mg). ◦ MRAs: Spironolactone (25–100 mg), Eplerenone (50–100 mg), Finerenone (10–20 mg). ◦ Amiloride/Triamterine: Amiloride (5–10 mg), Triamterine (50–100 mg).
  3. Resistant Hypertension Management (Flowchart 1):Step 1: Confirmation of Resistance ◦ If BP ≥130/80 mmHg on triple drug therapy (including a diuretic) OR ≥140/90 mmHg on quadruple therapy, and non-ordered treatments are excluded → Confirmed Resistant Hypertension. • Step 2: Exclusion of Secondary Causes ◦ Evaluate for: Lifestyle factors, OSA, Medication-induced hypertension, Anatomic causes (renal parenchymal disease, primary aldosteronism, renovascular disease, pregnancy disorders). ◦ If a secondary cause is found → Follow specific treatment path for that condition. • Step 3: Management Escalation (if no secondary cause identified) ◦ Step 1: Reaffirm adherence to lifestyle and medication. ◦ Step 2: Ensure full dosage of current medications. ◦ Step 3: Add Mineralocorticoid Receptor Antagonist (MRA). ◦ Step 4: Add vasodilator (e.g., hydralazine or potent minoxidil). • Step 4: Final Decision Point ◦ If BP remains uncontrolled after 6 months of intensive treatment → Consider antidromal secondary cause detection OR evaluation for surgical intervention.

PROGNOSIS & COMPLICATIONS

Cardiovascular Risk: ◦ Hypertension is a leading risk factor for stroke, CHD, HF, PAD, CKD/ESKD, and dementia. • Risk Quantification: ◦ Over 30-fold difference in 10-year predicted risk for patients with multiple risk factors vs. isolated hypertension.


SPECIAL CONSIDERATIONS

White Coat & Masked Hypertension: ◦ White coat: Lower risk profile; may be managed with nonpharmacologic interventions. ◦ Masked: Higher risk profile; requires pharmacologic therapy. • Secondary Hypertension: ◦ High suspicion in patients with new-onset, rapid worsening, or disproportionate organ damage.


KEY PEARLS & CLINICAL TRAPS

Measurement Accuracy: Ensure feet are flat on the floor to avoid overestimation. • Potassium Warning: Never supplement potassium in patients with advanced kidney disease or hyperkalemia. • Sodium Impact: A 25% reduction in sodium intake can lower SBP by ~5 mmHg. • Resistant Hypertension: If not controlled after 6 months of intensive treatment (including MRA/vasodilators), investigate for antidromal causes or surgery.


Reference Tables

TABLE 288-1 American College of Cardiology/American Heart Association Blood Pressure Classification System in Adults BP…

Harrison's 22e, p.2134

BP CATEGORY SYSTOLIC BP,
mmHg
DIASTOLIC BP,
mmHg
Normal BP <120 and <80
120–129 and
Stage 1 hypertension 130–139 or 80–89
≥140 or

TABLE 288-2 Summary Information for the Six Best Proven Nonpharmacologic Interventions That Lower Blood Pressure…

Harrison's 22e, p.2137

CHARACTERISTIC BP ASSOCIATION INTERVENTION REGIMEN EXPECTED CHANGE IN BP
Diet BPs are lower in those who
consume heart-healthy diets.
Consumption of a heart-
healthy diet, especially the
Dietary Approaches to Stop
Hypertension (DASH) diet.
DASH diet meal plans are readily
available.
With good adherence to the DASH
diet plan, an average SBP reduction
of about 5 mmHg can be expected in
patients with and 2–3 mmHg in those
without hypertension.
Increased body weight is
directly associated with SBP/
DBP. Almost 75% of U.S. adults
are overweight (BMI 25–<30 kg/
m2; 31%) or obese (BMI ≥30 kg/
m2; 42%).
In most patients, weight
loss should be achieved by
behavioral counseling. In a
minority, drug therapy should be
considered. Bariatric surgery
should be confined to adults with
a BMI ≥40 kg/m2 or ≥35 kg/m2 and
hypertension or another obesity-
related comorbidity.
Behavioral interventions are
aimed at a combination of calorie
reduction and increased physical
activity.
Sodium intake Dietary sodium is directly
associated with SBP, and
excessive intake may be
important in age-related
increases in BP.
Behavioral counseling, use of
salt substitutes, public health
messaging, and policy to reduce
the amount of sodium added
during food processing and
commercial preparation.
The sodium-BP dose response is
almost linear, so any reduction in
sodium intake is beneficial. Optimal
target recommendations vary from
<1500 to 2300 mg sodium intake
per day.
In high-quality behavior change trials,
average sodium intake is reduced
by ~25% and SBP by ~5 mmHg and
2–3 mmHg for adults with and without
hypertension, respectively. Use of salt
substitutes has resulted in prevention
of stroke (14%), CVD (13%), and all-
cause mortality (12%) in addition to BP
lowering.
Potassium intake is inversely
associated with BP and a lower
risk of CVD, especially stroke.
Dietary or pill supplementation,
but the former is preferred. An
additional intake of 2000 mg/d
(~50 mmol/d).
The recommended intake of
potassium in adults is ~3500 mg/d.
A dose-response meta-analysis
suggests supplementing
usual intake by ~1200 mg/d
may be optimal. Potassium
supplementation is contraindicated
in patients with hyperkalemia or
advanced kidney disease or who
are taking medicines that increase
the risk of hyperkalemia.
Physical activity Observational studies identify
a strong inverse association
between physical activity and
BP, hypertension, and CVD.
Aerobic, dynamic resistance,
and isometric resistance
exercises.
Most trials have evaluated aerobic
exercise interventions, and this
type of exercise is commonly
prescribed. Most guidelines
recommend ≥150 min of aerobic
exercise per week. However, any
increase in physical activity and
any type of exercise are likely to be
be beneficial.
In adults with hypertension, aerobic
exercise is likely to reduce SBP by
~5 mmHg and, when combined with
dynamic resistance exercise, by
~7 mmHg. The extent of BP lowering
depends on the starting level of BP and
success of the intervention.
Alcohol intake is associated
with BP and hypertension in
a direct and roughly linear
fashion, with no threshold for
the association.
Most commonly, substitution of
lower alcoholic or nonalcoholic
beverages, but behavioral
counseling and abstinence,
without access to alcohol, have
been used in some trials.
Most interventions have targeted a
reduction in alcohol consumption,
often to ≤2 and ≤1 standard drinks
per day in men and women,
respectively. However, any
reduction in alcohol consumption
is helpful.

TABLE 288-4 Summary Information for the Major Antihypertensive Drug Classes Used to Manage High Blood Pressure CLASS…

Harrison's 22e, p.2144

CLASS EXAMPLES DOSE
RANGE, mg
DOSE
FREQUENCY/D
METHOD OF ACTION SBP REDUCTION
VERSUS
PLACEBO, mmHg
(APPROXIMATE)
MOST COMMON
ADVERSE
EFFECTS
FREQUENCY
OF ADVERSE
EVENTS
COMMENTS
First-Step Drugs
Diuretics
(thiazide and
thiazide-like).
Chlorthalidone
Hydrochlorothiazide
Indapamide
12.5–25
12.5–50
1.25–2.5
1
1
1
Block sodium
reabsorption in distal
convoluted tubule
12 Hyponatremia
Hypokalemia
Hypercalcemia
Hyperuricemia
Hyperglycemia
Dyslipidemia
Similar to
placebo
Chlorthalidone
preferred due to
long half-life and
use in most U.S.
trials
Enalapril
Lisinopril
Benazepril
Ramipril
Trandolapril
5–40
10–40
10–40
2.5–20
1–4
1 or 2
1
1 or 2
1
1
Inhibit ACE
activity and Ang II
production
12.0 Cough
Hyperkalemia in
CKD
AKF in severe
bilateral RAS
Hypotension
Angioedema
Similar to
placebo
ARB Losartan
Valsartan
Azilsartan
Candesartan
Olmesartan
50–100
80–320
40–80
8–32
20–40
1 or 2
1
1
1
1
Block Ang II binding
to Ang receptors
12 Hyperkalemia in
CKD
Reduced GFR
AKF in severe
bilateral RAS
Hypotension
Similar to
placebo
Do not use in
combination with
ACE inhibitor;
contraindicated
in pregnancy
Amlodipine
Felodipine
Nifedipine LA
2.5–10
2.5–10
30–90
1
1
1
Blocks calcium
from entering cells,
primarily inhibiting
vasoconstriction
10 Peripheral edema
(dose-dependent)
Gingival
hyperplasia
Similar to
placebo
CCB
(non-DHP)
Diltiazem ER
Verapamil ER
120–360
100–300
1
1
(evening
administration
recommended)
Blocks calcium
from entering cells,
reducing heart rate
and vasoconstriction
9.0 Bradycardia
Nausea
Constipation.
Similar to
placebo
Do not use in
combination with
β-blockers
Do not use in
HFrEF or in high-
grade AV or SA
block
Other Drugs
Metoprolol
Carvedilol
Nebivolol
50–200
20–80
5–40
1
1
1
Block β-adrenergic
receptors
10 Asthma
Bradycardia
Fatigue
Exercise
intolerance
Impaired
concentration
Similar to
placebo
MRA Spironolactone
Eplerenone
Finerenone
25–100
50–100
10–20
1
2
1
Block distal tubule
mineralocorticoid
receptor activity
9 Hyperkalemia,
especially in
CKD and with
potassium-
sparing agents
Spironolactone
may cause
tender breasts,
gynecomastia,
and erectile
dysfunction in
men
9–20% Especially useful
in resistant
hypertension and
low-renin states
Amiloride
Triamterene
5–10
50–100
1 or 2
1 or 2
Block distal tubule
sodium reabsorption
NA Hyperkalemia,
especially in
CKD or with
other agents that
favor potassium
retention
Similar to
placebo