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Candidiasis

Part 5 | Infectious Diseases: Fungal · Part 5 – Infectious Diseases: Fungal · Chapter 222


Key Clinical Points

  1. C. auris is an emerging multidrug-resistant pathogen designated an urgent threat by CDC (2019) and critical priority by WHO (2022).
  2. Echinocandins are the first choice of treatment for disseminated candidiasis.
  3. C. auris exhibits unique tolerance to high temperature (up to 42°C) and saline concentrations (10%).
  4. Candidemia treatment duration is 2 weeks beyond the last positive blood culture and resolution of signs and symptoms.
  5. CMC is associated with monogenic disorders of IL-17 receptor signaling (e.g., STAT1, IL-17RA/RC mutations).
  6. C. auris strains can be resistant to all three major antifungal classes (azoles, echinocandins, polyenes).
  7. Recovery of Candida from sputum, urine, or peritoneal catheters may indicate mere colonization rather than deep-seated infection.
  8. The presence of ocular or macronodular skin lesions is highly suggestive of widespread infection of multiple deep organs.
  9. For C. auris, echinocandin treatment continues until sensitivities are determined.
  10. C. auris was first identified in 2009 from the ear drainage of a patient with an ear infection in Japan.

1. DEFINITION & OVERVIEW

Overview: The genus Candida encompasses >150 species, only a few of which cause disease in humans. ◦ Pathogenic species include: C. albicans, C. guilliermondii (Meyerozyma guilliermondii), C. krusei (Pichia kudriavzevii), C. parapsilosis, C. tropicalis, C. lusitaniae (Clavispora lusitaniae), C. dubliniensis, C. glabrata (Nakaseomyces glabratus), and C. auris. ◦ Morphology: Small, thin-walled yeast; reproduces by budding; exists as blastospores, pseudohyphae, and hyphae. ◦ Identification: Via biochemical testing or special agar (e.g., CHROMagar).

C. auris Status: Designed an urgent threat by CDC (2019) and critical priority by WHO (2022). ◦ Reasons: Multidrug resistance, persistence on surfaces, and association with high mortality.

1.1 Species Classification

Identified Pathogens: - C. albicans - C. guilliermondii (Meyerozyma guilliermondii) - C. krusei (Pichia kudriavzevii) - C. parapsilosis - C. tropicalis - C. lusitaniae (Clavispora lusitaniae) - C. dubliniensis - C. glabrata (Nakaseomyces glabratus) - C. auris


2. EPIDEMIOLOGY

Clinical Context: Common commensals that have become leading nosocomial pathogens in developed countries. ◦ U.S. Status: One of the four most common bloodstream pathogens in hospitalized patients.

C. auris Profile: First identified in 2009 (Japan); spread to >50 countries. ◦ Risk factors for high mortality (30–60% crude rate): Multidrug resistance, persistence on surfaces.

Risk Factors for Disseminated Candidiasis: (See Table 222-1) - Antibacterial agents - Indwelling intravenous catheters - Hyperalimentation fluids - Indwelling urinary catheters - Parenteral glucocorticoids - Severe burns - CARD9 deficiency (central nervous system) - Abdominal and thoracic surgery - Cytotoxic chemotherapy - Immunosuppressive agents for organ transplantation - Respirators - Myeloperoxidase deficiency - Neutropenia - Low birth weight (neonates) - Diabetes


3. ETIOLOGY & PATHOPHYSIOLOGY

Infection Mechanism: Occurs via hematogenous dissemination from mucosal surfaces or catheter sites. ◦ Tissue Invasion: Facilitated by pseudohyphae/hyphae formation (Note: C. glabrata and C. auris can cause infection without this morphology).

Virulence Factors: - Adhesins (e.g., candidalysin) - Biofilm formation

C. auris Unique Traits: - Tolerance to 42°C and 10% saline - Abiotic surface affinity (plastics, medical devices) - Persistent colonization capabilities - SCF1 adhesin (biofilm formation, virulence) - Aggregate-forming properties (immune evasion)


4. CLINICAL FEATURES

Mucocutaneous Infections: - Oral thrush: White adherent patches - Vulvovaginal candidiasis: Pruritus, curd-like discharge - Skin infections: Paronychia, intertrigo

Deeply Invasive Candidiasis: May involve the brain, heart, kidneys, or joints.

High-Suspicion Signs: - Ocular involvement (chorioretinitis) - Macronodular skin lesions (highly suggestive of hematogenously disseminated candidiasis; see Figure 222-2).

Chronic Mucocutaneous Candidiasis (CMC): Definition: A heterogeneous infection of the hair, nails, skin, and mucous membranes that persists despite intermittent antifungal therapy. - Onset: Infancy or first two decades of life - Immunologic dysfunction: IL-17 signaling defects (STAT1, IL-17RA/RC mutations) - Associated endocrine abnormalities: Hypoparathyroidism, adrenal insufficiency, autoimmune thyroiditis, alopecia, pernicious anemia


5. DIFFERENTIAL DIAGNOSIS

Colonization vs. Infection: Distinguishing these is critical for management. - Sputum/urine/peritoneal catheters: Presence of Candida likely indicates colonization rather than deep infection. - Blood cultures from patients with indwelling catheters: May reflect seeding, not dissemination.

C. auris Identification Challenges: May be misdiagnosed as other species due to atypical morphology (budding yeast, no germ tubes) and growth at 42°C on CHROMagar.


6. DIAGNOSTIC APPROACH

Microscopic Identification: - Identify pseudohyphae/hyphae on Gram stain or PAS stain.

Molecular & Specialized Methods: - MALDI-TOF MS and molecular techniques confirm C. auris (Table 222-2).

Decision-Making Steps for C. auris (Table 222-2): 1. Bruker Biotyper MALDI-TOF: - RUO libraries → C. auris confirmed - CA System library → C. auris confirmed 2. bioMérieux VITEK MS MALDI-TOF: - RUO library → C. auris - IVD library (v3.2) → C. auris - Older IVD libraries → C. haemulonii, C. lusitaniae, or No identification3. VITEK 2 YST: - Software version 8.01: - C. auris → C. auris confirmed - C. haemulonii / C. duobushaemulonii / Not identified → C. auris possible; Needs further workup - Older versions → C. auris possible; Needs further workup 4. API 20C: - Rhodotorula glutinis (if characteristic red color absent), C. sake, or Not identified 5. API ID 32C: - C. intermedia, C. sake, S. kluyveri → C. auris possible; Needs further workup 6. BD Phoenix: - C. catenulata, C. haemulonii, or Not identified 7. MicroScan: - No hyphal growth present + (C. lusitaniae/guilliermondii/parapsilosis) → Rule out those 3; C. auris possible; Needs further workup - Hyphal growth present → Possibly C. lusitaniae, C. guilliermondii, C. parapsilosis, or C. auris; Needs further workup - C. famata → C. auris possible; Needs further workup 8. RapID Yeast Plus: - C. parapsilosis → Test on corneal agar 9. GenMark ePlex BCID-FP Panel: - C. auris → C. auris confirmed


7. MANAGEMENT & TREATMENT

First-Line Therapy: Echinocandins are the first choice for disseminated candidiasis.

Treatment Duration: 1. Standard: 2 weeks beyond last positive blood culture and resolution of symptoms. 2. C. auris specific: Echinocandin treatment continues until sensitivities are determined.

Antifungal Selection by Species: - Non-auris strains → Azoles. - Auris strains → Echinocandins (monitor for resistance).

Mucocutaneous Treatment (Table 223): 1. Cutaneous: - Preferred: Topical azole. - Alternatives: Topical nystatin; Oral fluconazole (150 mg) or ibrexafungerp (300 mg twice daily for 1 day). 2. Oral (thrush): - Preferred: Fluconazole tablets (100–200 mg/d). - Alternatives: Clotrimazole troches, nystatin; or itraconazole solution (200 mg/d).

Systemic Agents for Disseminated Candidiasis (Table 224): 1. Amphotericin B deoxycholate: - Route: IV only. - Dose: 0.5–1.0 mg/kg daily (mostly replaced by lipid formulations) or 3.0–5.0 mg/kg daily. 2. Posaconazole: - Route: IV and oral. - Dose: 300 mg/d (IV); 200 mg tid (oral). Approved for prophylaxis. 3. Fluconazole: - Route: IV and oral. - Dose: 400 mg/d. Most commonly used. 4. Voriconazole: - Route: IV and oral. - Dose: 400 mg/d. (Note: multiple drug interactions, visual hallucinations, fluorosis, phototoxicity; approved for candidemia in nonneutropenic patients). 5. Isavuconazole: - Route: IV only. - Dose: 50 mg/d. 6. [Additional Agent]: - Route: IV only. - Dose: 100 mg/d.

Echinocandin Doses for C. auris (Table 226): 1. Caspofungin: - Adults: Loading dose 70 mg IV, then 50 mg IV daily. - Children (>2 months): Loading dose 70 mg/m² per day IV, then 50 mg/m² per day IV. - Infants (<2 months): 25 mg/m² per day IV. 2. Micafungin: - Adults: 100 mg IV daily. - Children (>2 months): 2 mg/kg per day IV (option to increase to 4 mg/kg per day in children ≥ 40 kg). - Infants (<2 months): 10 mg/kg per day IV.


8. PROGNOSIS & COMPLICATIONS

C. auris Mortality: 30–60% crude mortality rate.

Urgent Interventions: - Endophthalmitis: Requires prompt intervention (vitrectomy, intraocular antifungals).

Prognostic Factors: - Timely diagnosis. - Appropriate antifungal therapy. - Effective infection control measures.


9. SPECIAL CONSIDERATIONS

Neonates: - Risk factors: Low birth weight, prolonged hospitalization. - Clinical features: Generalized cutaneous eruptions, candidemia.


10. KEY PEARLS & CLINICAL TRAPS

C. auris Identification: May be misidentified as other species due to atypical morphology (budding yeast, no hyphal growth). • Clinical Red Flags: Ocular or macronodular skin lesions strongly suggest hematogenously disseminated candidiasis. • Colonization Trap: Candida in sputum, urine, or peritoneal catheters may reflect colonization, not infection. • First-line Choice: Echinocandins are preferred for C. auris despite potential resistance. • Treatment Duration: Standard is 2 weeks post-clearance; however, C. auris requires continued echinocandin until sensitivities are known.


Reference Tables

TABLE 222-1 Well-Recognized Factors and Conditions Predisposing to Hematogenously Disseminated Candidiasis…

Harrison's 22e, p.1707

Antibacterial agents
Indwelling intravenous catheters
Hyperalimentation fluids
Indwelling urinary catheters
Parenteral glucocorticoids
Severe burns
CARD9 deficiency (central nervous
system)
Abdominal and thoracic surgery
Cytotoxic chemotherapy
Immunosuppressive agents for organ
transplantation
Respirators
Myeloperoxidase deficiency
Neutropenia
Low birth weight (neonates)
Diabetes

TABLE 222-2 Typical Decision-Making Steps in the Diagnosis of C. auris NO. 1. 2.

Harrison's 22e, p.1710

NO. METHOD DATABASE/SOFTWARE INITIAL FINDING CONFIRMATION
1. Bruker Biotyper
MALDI-TOF
RUO libraries C. auris C. auris
CA System library C. auris C. auris
bioMérieux VITEK
MS MALDI-TOF
RUO library C. auris
IVD library (v3.2) C. auris
Older IVD libraries C. haemulonii
C. lusitaniae
No identification
3. VITEK 2 YST Software version 8.01 C. auris C. auris confirmed
C. haemulonii C. auris possible: Needs further workup
C. duobushaemulonii C. auris possible: Needs further workup
Candida spp. not identified C. auris possible: Needs further workup
Older versions C. haemulonii C. auris possible: Needs further workup
C. duobushaemulonii C. auris possible: Needs further workup
Candida spp. not identified C. auris possible: Needs further workup
API 20C Rhodotorula glutinis, if characteristic red color absent
C. sake
Candida spp. not identified
5. API ID 32C C. intermedia C. auris possible: Needs further workup
C. sake C. auris possible: Needs further workup
Saccharomyces kluyveri C. auris possible: Needs further workup
BD Phoenix C. catenulata
C. haemulonii
Candida spp. not identified
7. MicroScan C. lusitaniae No hyphal growth present Can rule out C. lusitaniae, C.
guilliermondii, and C. parapsilosis.
C. auris possible: Needs further workup
C. guilliermondii
C. parapsilosis
C. lusitaniae Hyphal growth present Possibly C. lusitaniae,
C. guilliermondii, C. parapsilosis, or
C. auris: Needs further workup
C. guilliermondii
C. parapsilosis
C. famata C. auris possible: Needs further workup
Candida spp. not identified C. auris possible: Needs further workup
RapID Yeast Plus C. parapsilosis → Test on
corneal agar
No hyphal growth present
Hyphal growth present
Candida spp. not identified
9. GenMark ePlex
BCID-FP Panel
C. auris C. auris confirmed

TABLE 222-3 Treatment of Mucocutaneous Candidal Infections DISEASE Cutaneous Vulvovaginal

Harrison's 22e, p.1710

DISEASE PREFERRED TREATMENT ALTERNATIVES
Cutaneous Topical azole Topical nystatin
Oral fluconazole (150 mg) or
ibrexafungerp (300 mg twice daily for 1
day) or azole cream or suppository
Oral (thrush) Fluconazole tablets (100–200 mg/d) Clotrimazole trashes,
nystatin
Fluconazole tablets (100–200 mg/d) or
itraconazole solution (200 mg/d)

TABLE 222-4 Agents for the Treatment of Disseminated Candidiasis AGENT Amphotericin B deoxycholate Amphotericin B lipid…

Harrison's 22e, p.1711

AGENT ROUTE OF
ADMINISTRATION
DOSEa COMMENT
Amphotericin B deoxycholate IV only 0.5–1.0 mg/kg daily Mostly replaced by lipid formulations
IV only
IV only
IV only
3.0–5.0 mg/kg daily
3.0–5.0 mg/kg daily
3.0–5.0 mg/kg daily
Azolesb
Posaconazole IV and oral 300 mg/d (IV)
200 mg tid (oral)
Approved for prophylaxis
Fluconazole IV and oral 400 mg/d Most commonly used
Voriconazole IV and oral 400 mg/d Multiple drug interactions, visual hallucinations, fluorosis, phototoxicity
Approved for candidemia in nonneutropenic patients
IV only
IV only
IV only
IV only
50 mg/d
100 mg/d
100 mg/d
400 mg loading dose, 200 mg
once weekly thereafter

TABLE 222-5 Typical MICs of Available Antifungal Drugs for C. auris DRUG Amphotericin B Fluconazole Itraconazole…

Harrison's 22e, p.1711

DRUG TENTATIVE
RESISTANCE
BREAKPOINTSa
MIC RANGE, μg/mL
MIC MIC
50
MIC
90
Amphotericin B ≥2 0.06–8 0.5–1 2–4
≥32 0.12–≥64 ≥64
Itraconazole N/A 0.032–2 0.06–0.5 0.25–1
N/A 0.032–16 0.5–2
Posaconazole N/A 0.015–16 0.016–0.5 0.125–2
N/A 0.015–4 0.125–0.25
Caspofungin ≥2 0.03–16 0.25–1 1–2
≥4 0.015–16 0.125–0.5
Micafungin ≥4 0.015–8 0.125–0.25 0.25–2

Harrison's 22e, p.1712

DRUG ADULTS CHILDREN
(>2 MONTHS)
INFANTS
(<2 MONTHS)
Caspofungin Loading dose
70 mg IV, then
50 mg IV daily
Loading dose 70 mg/m2 per
day IV, then 50 mg/m2 per
day IV
25 mg/m2 per
day IV
Loading dose
200 mg IV, then
100 mg IV daily
Not approved for use in
children
Micafungin 100 mg IV daily 2 mg/kg per day IV with
option to increase to 4 mg/
kg per day IV in children at
least 40 kg
10 mg/kg per
day IV