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Breast Cancer

Chapter 84 | Breast Cancer

KEY CLINICAL POINTS

  • Breast cancer is the most common nonskin cancer in women, with ~310,000 new cases in the U.S. in 2024 and a 5-year relative survival rate of 91%.
  • Key risk factors include female gender, age >55, family history, BRCA mutations, and hormone replacement therapy.
  • Staging is based on AJCC TNM criteria, with survival rates varying by stage (Stage I: >99%, Stage IV: 29%).
  • Treatment is tailored by molecular subtypes (Luminal A/B, HER2+, TNBC) and includes surgery, endocrine therapy, targeted agents, and chemotherapy.
  • Metastatic breast cancer is managed with systemic therapies, including CDK4/6 inhibitors, PARP inhibitors, and immunotherapy.

1. DEFINITION & OVERVIEW

Breast cancer is a malignant neoplasm arising from epithelial cells in the breast. It is classified as in situ (DCIS/LCIS) or invasive, with subtypes defined by receptor status (ER/PR/HER2) and molecular profiles.

Table 84-1: Five-Year Breast Cancer Relative Survival Rate (%) by Stage and Race/Ethnicity

STAGE ALL WHITE BLACK AIAN HISPANIC API
I >99 >99 >99 >99 >99 >99
II 93 93 89 93 92 94
III 75 77 64 72 74 77
IV 29 31 20 35 29 34

1.1 Molecular Subtypes

Invasive breast cancer is categorized into four major subtypes: Luminal A (ER+/PR+/HER2-), Luminal B (ER+/PR+/HER2- with high proliferation), HER2-positive (ER-/PR-/HER2+), and Triple-Negative (ER-/PR-/HER2-).

1.2 Staging Systems

Staging follows AJCC TNM criteria (8th edition). Stage I: localized; Stage II: regional; Stage III: locally advanced; Stage IV: distant metastases (M1).

2. EPIDEMIOLOGY

Breast cancer is the most common cancer in women globally. In the U.S., ~310,000 new cases in 2024, with ~42,250 deaths. Incidence peaks in non-Hispanic whites, while mortality is highest in non-Hispanic Black women.

2.1 Risk Factors

Non-genetic: female gender, age >55, late menopause, early menarche, obesity (postmenopausal). Genetic: BRCA1/2 mutations, Li-Fraumeni syndrome, PALB2, ATM, PTEN mutations.

2.2 Demographics

Incidence highest in non-Hispanic whites, lowest in Asian/Pacific Islanders. Mortality rates vary by race/ethnicity, with highest in non-Hispanic Black women.

3. ETIOLOGY & PATHOPHYSIOLOGY

Prolonged estrogen exposure drives most breast cancers. Mutations in DNA repair genes (BRCA1/2, TP53) lead to genomic instability. HER2 overexpression and hormone receptor dysregulation drive tumor growth.

3.1 Genetic Mutations

BRCA1/2 mutations confer 50–80% lifetime risk of invasive breast cancer and 30% risk of ovarian cancer. Other mutations (TP53, PALB2, ATM) increase risk in specific syndromes.

3.2 Hormonal Factors

Estrogen and progesterone receptor signaling promote proliferation. Hormone replacement therapy (HRT) increases risk, while postmenopausal obesity exacerbates estrogen exposure.

4. CLINICAL FEATURES

Common presentations include breast mass, skin changes, nipple retraction, or axillary lymphadenopathy. Metastases may occur in bones, lungs, liver, or brain.

4.1 In Situ vs. Invasive

DCIS (ductal carcinoma in situ) is confined to ducts; LCIS (lobular carcinoma in situ) is a marker for future invasive disease.

4.2 Metastatic Patterns

Common sites: bones, lungs, liver, brain. Metastatic disease may present with bone pain, pleural effusion, or neurological symptoms.

5. DIFFERENTIAL DIAGNOSIS

Differential includes fibroadenoma, fibrocystic changes, mastitis, and gynecomastia. Imaging (mammography, ultrasound) and biopsy are critical for distinction.

5.1 Benign Lesions

Fibroadenoma, cysts, and intraductal papilloma may mimic malignancy on imaging.

5.2 Inflammatory Breast Cancer

Distinctive presentation with erythema, swelling, and skin dimpling due to lymphatic invasion.

6. INVESTIGATIONS & DIAGNOSIS

Diagnosis combines imaging (mammography, ultrasound, MRI), biopsy, and biomarker testing (ER, PR, HER2). Staging includes CT, PET, and bone scans for metastases.

Table 84-1: Five-Year Breast Cancer Relative Survival Rate (%) by Stage and Race/Ethnicity

STAGE ALL WHITE BLACK AIAN HISPANIC API
I >99 >99 >99 >99 >99 >99
II 93 93 89 93 92 94
III 75 77 64 72 74 77
IV 29 31 20 35 29 34

6.1 Diagnostic Workup

Suspicious findings undergo imaging (mammography, ultrasound, MRI) followed by core biopsy. MRI is more sensitive than mammography for dense breasts.

6.2 Biomarker Testing

ER, PR, HER2 status and genomic assays (Oncotype DX, MammaPrint) guide treatment decisions.

7. MANAGEMENT & TREATMENT

Treatment is tailored by subtype and stage. Surgery (lumpectomy/mastectomy), radiation, endocrine therapy, targeted agents, and chemotherapy are core modalities.

Table 84-1: Five-Year Breast Cancer Relative Survival Rate (%) by Stage and Race/Ethnicity

7.1 Early-Stage Management

Breast-conserving therapy (lumpectomy + radiation) or mastectomy. Endocrine therapy (tamoxifen, aromatase inhibitors) for ER+/PR+ tumors.

7.2 Metastatic Disease

Systemic therapies include CDK4/6 inhibitors (abemaciclib, ribociclib), PARP inhibitors (olaparib), and immunotherapy (pembrolizumab for TNBC).

8. PROGNOSIS & COMPLICATIONS

5-year survival >99% for Stage I, 29% for Stage IV. Complications include cardiotoxicity (from anthracyclines), osteoporosis (from aromatase inhibitors), and lymphedema.

8.1 Survival Rates

Stage I: >99%, Stage II: 93%, Stage III: 75%, Stage IV: 29% (U.S., 2012–2018).

8.2 Long-Term Effects

Cardiotoxicity, osteoporosis, lymphedema, and cognitive dysfunction may occur with treatment.

9. SPECIAL CONSIDERATIONS

Management in pregnancy requires balancing maternal and fetal safety. Fertility preservation is critical for premenopausal women. Germline testing is recommended for BRCA carriers.

9.1 Pregnancy

Surgery safe in second/third trimesters; radiation delayed until after delivery. Chemotherapy (e.g., doxorubicin) may be used in second/third trimesters.

9.2 Fertility Preservation

Ovarian suppression with GnRH agonists or cryopreservation of oocytes recommended for premenopausal women.

10. KEY POINTS & CLINICAL PEARLS

  1. Breast cancer is the most common cancer in women, with 5-year survival >91% in the U.S. 2. Staging guides treatment and prognosis. 3. Molecular subtypes dictate therapy (e.g., HER2-targeted agents for HER2+ tumors). 4. Endocrine therapy is foundational for ER+/PR+ tumors. 5. Metastatic disease requires systemic therapy with CDK4/6 inhibitors or PARP inhibitors.