Skip to content

Approach to Articular and Musculoskeletal Disorders

Chapter 382 | Part 11: Immune-Mediated, Inflammatory, and Rheumatologic Disorders

KEY CLINICAL POINTS

  • Distinguish between articular (joint) and nonarticular (periarticular) musculoskeletal disorders using pain characteristics, range of motion, and swelling.
  • Red flag conditions include septic arthritis, gout, and fractures, requiring urgent evaluation.
  • Inflammatory disorders (e.g., RA, gout) present with swelling, warmth, and systemic symptoms, while noninflammatory conditions (e.g., OA, fibromyalgia) lack these features.
  • Use the algorithm in Figure 382-1 to guide differential diagnosis based on chronology, pathology, and systemic features.
  • Synovial fluid analysis (via polarized microscopy) is critical for diagnosing crystal-induced arthritis (gout/pseudogout) and infection.

1. DEFINITION & OVERVIEW

Musculoskeletal disorders encompass articular (joint) and nonarticular (periarticular) conditions. Articular structures include synovium, cartilage, and ligaments, while nonarticular structures involve tendons, bursae, and muscles. Nonarticular pain is more common and may mimic arthritis. Key features include deep pain with swelling in articular disorders vs. focal tenderness in nonarticular conditions.

Table 382-1: Evaluation of Patients with Musculoskeletal Complaints

Goal Approach
Accurate diagnosis Determine chronology (acute vs. chronic)
Timely provision of therapy Assess inflammatory vs. noninflammatory process
Avoidance of unnecessary testing Anatomic localization (articular vs. nonarticular)
Identification of red flags Determine extent of involvement (monoarticular, polyarticular)

1.1 Articular vs. Nonarticular Disorders

Articular disorders: Deep pain, limited range of motion, swelling, crepitus. Nonarticular disorders: Focal tenderness, pain with active motion, no swelling. Nonarticular conditions (e.g., tendinitis, fibromyalgia) often mimic arthritis.

1.2 Red Flag Conditions

Urgent evaluation is needed for septic arthritis, acute crystal-induced arthritis (gout/pseudogout), and fractures. These present with acute onset, monoarticular pain, and systemic symptoms.

2. EPIDEMIOLOGY

Musculoskeletal disorders affect 1 in 3 U.S. adults (127 million). 54.4 million have physician-diagnosed arthritis. Common conditions include OA (50% of adults >65), RA (1%), gout (men), and fibromyalgia (2-4% of women). Age-related risks: OA, osteoporosis, and fractures in elderly; gout and pseudogout in middle-aged men.

2.1 Demographics

OA: 15-25% of outpatient visits. RA: 1% prevalence. Gout: 3.9 million in U.S. (men > women). Fibromyalgia: 2-4% of women. PMR: 0.1-0.3% of adults >50.

2.2 Risk Factors

Trauma, repetitive strain, obesity, diabetes, renal insufficiency, and medications (e.g., corticosteroids) increase risk. Age >60: OA, osteoporosis, fractures. Men: gout, spondyloarthritis. Women: RA, osteoporosis.

3. ETIOLOGY & PATHOPHYSIOLOGY

Inflammatory disorders (infectious, crystal, autoimmune) vs. noninflammatory (trauma, degenerative, neoplastic). Crystal-induced arthritis (gout: monosodium urate; pseudogout: calcium pyrophosphate). Autoimmune: RA (synovitis), SLE (vasculitis). Noninflammatory: OA (cartilage degeneration), fibromyalgia (central pain amplification).

Table 382-2: Drug-Induced Musculoskeletal Conditions

Condition Associated Drugs
Arthralgias Quinidine, beta blockers, statins, HIV protease inhibitors
Myalgias/myopathy Glucocorticoids, penicillamine, statins, cyclosporine
Tendon rupture/tendinitis Quinolones, glucocorticoids, statins
Gout Diuretics, alcohol, fructose-containing soft drinks
Drug-induced lupus Hydralazine, procainamide, phenytoin

3.1 Crystal-Induced Arthritis

Gout: monosodium urate crystals (polarized microscopy: needle-shaped, negative birefringence). Pseudogout: calcium pyrophosphate dihydrate (rhomboid-shaped, positive birefringence).

3.2 Autoimmune Mechanisms

RA: T-cell mediated synovitis, pannus formation. SLE: autoantibodies (ANA, anti-dsDNA), vasculitis. Psoriatic arthritis: HLA-B27, enthesitis.

4. CLINICAL FEATURES

Acute vs. chronic presentation. Articular: swelling, warmth, limited motion. Nonarticular: focal tenderness. Inflammatory: prolonged morning stiffness, systemic symptoms (fever, weight loss). Noninflammatory: intermittent stiffness (<45 min), fatigue. Red flags: fever, rash, joint effusion.

4.1 Pain Characteristics

Articular: Deep, diffuse pain with swelling. Nonarticular: Focal, localized tenderness. Fibromyalgia: Widespread pain with no objective findings.

4.2 Systemic Symptoms

Inflammatory: Fever, weight loss, anemia. Noninflammatory: Fatigue, sleep disturbances. SLE: Rash, serositis. RA: Morning stiffness >30 min.

5. DIFFERENTIAL DIAGNOSIS

Articular: RA, gout, septic arthritis. Nonarticular: Fibromyalgia, tendinitis, bursitis. Crystal-induced: Gout (monoarticular), pseudogout (oligoarticular). Inflammatory: SLE, vasculitis. Noninflammatory: OA, PMR, myopathy.

5.1 Articular vs. Nonarticular

Articular: Swelling, warmth, limited motion. Nonarticular: Focal tenderness, no swelling. Fibromyalgia: Widespread pain with no objective findings.

5.2 Crystal-Induced Arthritis

Gout: Acute monoarticular pain, tophi, hyperuricemia. Pseudogout: Chronic, symmetric, calcification (chondrocalcinosis).

6. INVESTIGATIONS & DIAGNOSIS

Laboratory tests: ESR, CRP, uric acid, RF, ANA. Imaging: X-ray (OA, fractures), MRI (inflammation, necrosis), ultrasound (tendinitis, bursitis). Synovial fluid analysis: Polarized microscopy for crystals, Gram stain for infection. Algorithms: Figure 382-1 for differential diagnosis.

Table 382-4: Antinuclear Antibody (ANA) Patterns

ANA Pattern Antigen Clinical Correlate
Diffuse Deoxyribonucleoprotein Nonspecific
Peripheral (rim) ds-DNA 50% SLE
Speckled U1-RNP MCTD
Centromere Kinetochore CREST syndrome

6.1 Laboratory Tests

ESR/CRP: Inflammatory markers. Uric acid: Gout diagnosis. RF/ANA: Autoimmune disorders. CBC: Infection, anemia.

6.2 Imaging

X-ray: OA, fractures. MRI: Inflammation, necrosis. Ultrasound: Soft tissue abnormalities. CT: Spinal stenosis, fractures.

7. MANAGEMENT & TREATMENT

Acute: NSAIDs, colchicine (gout), antibiotics (infection). Chronic: DMARDs (RA), biologics (TNF inhibitors), corticosteroids. Nonpharmacologic: Physical therapy, weight management, exercise. Surgical: Joint replacement, arthroscopy for meniscal tears.

7.1 Acute Inflammatory Arthritis

Gout: Colchicine, NSAIDs, corticosteroids. Septic arthritis: IV antibiotics, arthroscopy. Pseudogout: NSAIDs, corticosteroids.

7.2 Chronic Conditions

RA: Methotrexate, TNF inhibitors. OA: NSAIDs, hyaluronic acid injections. Fibromyalgia: Pregabalin, CBT.

8. PROGNOSIS & COMPLICATIONS

Inflammatory disorders (RA, SLE): Risk of joint destruction, comorbidities (cardiovascular, renal). Noninflammatory: Chronic pain, disability. Complications: Osteonecrosis (corticosteroids), infection (immunosuppression), drug-induced myopathy.

8.1 Long-Term Outcomes

RA: Erosive joint damage without treatment. SLE: Renal failure, cardiovascular disease. Gout: Tophi, joint destruction.

8.2 Complications

Corticosteroid use: Osteonecrosis, myopathy. Immunosuppression: Infections, malignancy. Chronic pain: Depression, sleep disturbances.

9. SPECIAL CONSIDERATIONS

Pregnancy: Avoid NSAIDs, corticosteroids. Elderly: Osteoporosis, drug interactions. Pediatrics: Juvenile idiopathic arthritis, growth plate involvement. Telehealth: Virtual exams for range of motion, gait analysis.

9.1 Pregnancy

Avoid NSAIDs (third trimester), corticosteroids (risk of cleft palate). Use acetaminophen. Monitor for gestational hypertension.

9.2 Elderly Patients

OA, osteoporosis, fractures. Avoid NSAIDs (renal risk). Use low-dose corticosteroids. Monitor for drug interactions.

10. KEY POINTS & CLINICAL PEARLS

Use the algorithm in Figure 382-1 to guide diagnosis. Synovial fluid analysis is critical for crystal-induced arthritis. Distinguish between articular and nonarticular pain. Monitor uric acid levels in gout. Avoid NSAIDs in elderly patients. Telehealth can assess gait and range of motion.